Abstract
Hepatocellular adenoma (HCA) is a benign liver neoplasm composed of hepatocytes. We
experienced HCA demonstrating a high uptake of 18F-fluorodeoxyglucose (FDG) on positron emission tomography–computed tomography, mimicking
a malignant tumor. The mechanism underlying the uptake of FDG has not been identified.
Here, we discuss that an enhancement of glucose metabolism via an increased expression
of glucose transporter 2 may have a role in the high uptake of FDG shown by HCAs.
Keywords
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Article info
Publication history
Published online: June 12, 2014
Accepted:
June 2,
2014
Received in revised form:
May 19,
2014
Received:
March 18,
2014
Identification
Copyright
© 2014 Elsevier Inc. Published by Elsevier Inc. All rights reserved.
